NEWS AND FILLER
Year : 2012  |  Volume : 22  |  Issue : 1  |  Page : 70-71

Dyselectrolytemias: Word search

A Gupta, M Biyani
Division of Nephrology, University of Ottawa, Ottawa, Canada

Correspondence Address:
A Gupta
Unit 318, 1833 Riverside Drive, Ottawa, ON, K1G 0E8
Canada

Source of Support: None, Conflict of Interest: None

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DOI: 10.4103/0971-4065.86414

Clues

1. Proton pump inhibitors and epidermal growth factor receptor antagonists may cause this dyselectrolytemia.
2. Polyuria, hypokalemia, and hypomagnesemia occurring in this infective cause of acute kidney injury.
3. Cyclical excessive vomiting and compulsive taking of hot baths with this substance abuse.
4. Syndrome characterized by hypokalemic alkalosis, hypomagnesemia, and hypocalciuria.
5. This root extract may cause mineralocorticoid excess.
6. Possible adverse effect of bevacizumab.
7. Continuous renal replacement therapy causing low ionized calcium, high anion gap metabolic acidosis, and this toxicity in liver failure patients.
8. Electrolyte imbalance may induce this syndrome characterized by ST segment elevation in right precordial leads followed by negative T wave unrelated to ischemia on electrocardiogram.
9. Hyponatremia after the use of this alkylating agent.
10. Syndrome characterized by skin hyperpigmentation, polyostotic fibrous dysplasia, and hypophosphatemia.
11. Mutation in this gene along with elevated fibroblast growth factor (FGF)-23 causes X-linked hypophosphatemic rickets.
12. FGF-23, FGF-7, matrix extracellular phosphoglycoprotein, and secreted frizzled-related protein-4 are examples of .......onin.
13. Mutation in with-no-lysine kinases (WNK) 1 and 4 are responsible for this hyperkalemic syndrome.
14. Aldosterone-sensitive Cl ^- /HCO3 ^- exchanger in cortical collecting duct encoded by Slc26a4.
15. A cofactor mandatory for FGF-23 action.
16. Spurious hyperphosphatemia after the use of this locking agent in hemodialysis.
17. Imatinib causes renal wasting of this electrolyte.
18. Cause of monogenic hypercalciuric nephrolithiasis.
19. Autosomal recessive (AR) lysosomal storage disorder causing nephrogenic diabetes insipidus.
20. Antibiotic causing neurotoxicity and not so commonly hyponatremia.
21. Hyperchloremic metabolic acidosis and kidney stones with this antiseizure drug.
22. Pseudo-Bartter syndrome developing in AR disorder of Cl ^- channel defects.
23. Intestinal syndrome with hypokalemia.
24. Vitamin A intoxication and this dyselectrolytemia.
25. Angiotensin-converting enzyme inhibitors may cause this metabolic abnormality.
26. Antibiotic class associated with hypokalemia and metabolic alkalosis.
27. Dyselectrolytemia occurring by placing blood sample on ice and resultant urgent nephrology consult.
28. Acute severe oleander poisoning causing this dyselectrolytemia.
29. Be careful of potassium correction in this dyselectrolytemia.
30. Agents to enhance free water excretion.

This article has been cited by 1 Novel Educational Approaches to Enhance Learning and Interest in Nephrology Kenar D. Jhaveri,Matthew A. Sparks,Hitesh H. Shah Advances in Chronic Kidney Disease. 2013; 20(4): 336 [Pubmed] | [DOI]